ccd camera hisca model c6790 (Hamamatsu)
90
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Hamamatsu
ccd camera hisca model c6790
Ccd Camera Hisca Model C6790, supplied by Hamamatsu, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/ccd+camera+hisca+model+c6790/ccd+camera+c6790/pm31220549-92-39-44
Average 90 stars, based on 1 article reviews
Ccd Camera Hisca Model C6790, supplied by Hamamatsu, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/ccd+camera+hisca+model+c6790/ccd+camera+c6790/pm31220549-92-39-44
Average 90 stars, based on 1 article reviews
ccd camera hisca model c6790 - by Bioz Stars,
2026-10
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Modification:Article Title: Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells. Article Snippet: Chin T-Y, Lin H-C, Kuo J-P, Chueh S-H. Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells.. Am J Physiol Cell Physiol 292: C383–C395, 2007; doi:10.1152/ajpcell.00069.2006.—A sustained increase in the cytosolic Ca concentration ([Ca ]i) can cause cell death.. In this study, we found that, in cultured porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stress, triggered by depletion of Ca stores by thapsigargin (TG), induced an increase in the [Ca ]i and cell death. Article Title: Ultraviolet B irradiation induced Nrf2 degradation occurs via activation of TRPV1 channels in human dermal fibroblasts. Article Snippet: Ultraviolet (UV) irradiation causes cellular oxidative stress.. Under redox imbalance, Keap1-dependent Nrf2 degradation is minimal.. In this study, we examined the role of Ca in Nrf2 homeostasis after UVB irradiation using human dermal fibroblasts. Article Title: Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells. Article Snippet: Contents lists available at SciVerse ScienceDirect European Journal of Pharmacology j ourna l homepage: www.e lsev ie r .com/ locate /e jphar Molecular and Cellular Pharmacology Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells Kao-Chang Lin b,d, Pei-Shan Liu c, Pei-Yu Peng a, Sheau-Huei Chueh a,⁎ a Department of Biochemistry, National Defense Medical Center, Taipei, Taiwan, ROC b Department of Neurology, Chi Mei Medical Center, Tainan, Taiwan, ROC c Department of Microbiology, Soochow University, Shihlin, Taipei, Taiwan, ROC d Department of Biotechnology, Southern Taiwan University, Tainan, Taiwan, ROC ⁎ Corresponding author at: Department of Biochemis Center, 161, Section 6, Min-Chuan East Road, Taipei, Ta 0741; fax: +886 2 8792 3106.. E-mail addresses: gaujang@mail2000.com.tw (K.-C. pslediting@mail.scu.edu.tw (P.-S. Liu), lovelyfish92@yah shch@mail.ndmctsgh.edu.tw (S.-H. Chueh).. 0014-2999/$ – see front matter © 2012 Elsevier B.V. All doi:10.1016/j.ejphar.2012.04.015 a b s t r a c t a r t i c l e i n f o Article history: Received 7 March 2012 Received in revised form 4 April 2012 Accepted 5 April 2012 Available online 20 April 2012 Keywords: Porcine aortic smooth cell Autophagy Endoplasmic reticulum stress Phorbol ester Thapsigargin Extracellular signal-regulated kinase We have previously shown that, in porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stressor thapsigargin simultaneously activate the mitochondrial caspase-dependent death cascade and an extracellular signal-regulated kinase (ERK)-dependent pathway, which inhibits the caspase-independent death pathway. Fluorescence:Article Title: Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells. Article Snippet: Chin T-Y, Lin H-C, Kuo J-P, Chueh S-H. Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells.. Am J Physiol Cell Physiol 292: C383–C395, 2007; doi:10.1152/ajpcell.00069.2006.—A sustained increase in the cytosolic Ca concentration ([Ca ]i) can cause cell death.. In this study, we found that, in cultured porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stress, triggered by depletion of Ca stores by thapsigargin (TG), induced an increase in the [Ca ]i and cell death. Article Title: Ultraviolet B irradiation induced Nrf2 degradation occurs via activation of TRPV1 channels in human dermal fibroblasts. Article Snippet: Ultraviolet (UV) irradiation causes cellular oxidative stress.. Under redox imbalance, Keap1-dependent Nrf2 degradation is minimal.. In this study, we examined the role of Ca in Nrf2 homeostasis after UVB irradiation using human dermal fibroblasts. Article Title: Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells. Article Snippet: Contents lists available at SciVerse ScienceDirect European Journal of Pharmacology j ourna l homepage: www.e lsev ie r .com/ locate /e jphar Molecular and Cellular Pharmacology Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells Kao-Chang Lin b,d, Pei-Shan Liu c, Pei-Yu Peng a, Sheau-Huei Chueh a,⁎ a Department of Biochemistry, National Defense Medical Center, Taipei, Taiwan, ROC b Department of Neurology, Chi Mei Medical Center, Tainan, Taiwan, ROC c Department of Microbiology, Soochow University, Shihlin, Taipei, Taiwan, ROC d Department of Biotechnology, Southern Taiwan University, Tainan, Taiwan, ROC ⁎ Corresponding author at: Department of Biochemis Center, 161, Section 6, Min-Chuan East Road, Taipei, Ta 0741; fax: +886 2 8792 3106.. E-mail addresses: gaujang@mail2000.com.tw (K.-C. pslediting@mail.scu.edu.tw (P.-S. Liu), lovelyfish92@yah shch@mail.ndmctsgh.edu.tw (S.-H. Chueh).. 0014-2999/$ – see front matter © 2012 Elsevier B.V. All doi:10.1016/j.ejphar.2012.04.015 a b s t r a c t a r t i c l e i n f o Article history: Received 7 March 2012 Received in revised form 4 April 2012 Accepted 5 April 2012 Available online 20 April 2012 Keywords: Porcine aortic smooth cell Autophagy Endoplasmic reticulum stress Phorbol ester Thapsigargin Extracellular signal-regulated kinase We have previously shown that, in porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stressor thapsigargin simultaneously activate the mitochondrial caspase-dependent death cascade and an extracellular signal-regulated kinase (ERK)-dependent pathway, which inhibits the caspase-independent death pathway. Microscopy:Article Title: Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells. Article Snippet: Chin T-Y, Lin H-C, Kuo J-P, Chueh S-H. Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells.. Am J Physiol Cell Physiol 292: C383–C395, 2007; doi:10.1152/ajpcell.00069.2006.—A sustained increase in the cytosolic Ca concentration ([Ca ]i) can cause cell death.. In this study, we found that, in cultured porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stress, triggered by depletion of Ca stores by thapsigargin (TG), induced an increase in the [Ca ]i and cell death. Article Title: Ultraviolet B irradiation induced Nrf2 degradation occurs via activation of TRPV1 channels in human dermal fibroblasts. Article Snippet: Ultraviolet (UV) irradiation causes cellular oxidative stress.. Under redox imbalance, Keap1-dependent Nrf2 degradation is minimal.. In this study, we examined the role of Ca in Nrf2 homeostasis after UVB irradiation using human dermal fibroblasts. Article Title: Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells. Article Snippet: Contents lists available at SciVerse ScienceDirect European Journal of Pharmacology j ourna l homepage: www.e lsev ie r .com/ locate /e jphar Molecular and Cellular Pharmacology Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells Kao-Chang Lin b,d, Pei-Shan Liu c, Pei-Yu Peng a, Sheau-Huei Chueh a,⁎ a Department of Biochemistry, National Defense Medical Center, Taipei, Taiwan, ROC b Department of Neurology, Chi Mei Medical Center, Tainan, Taiwan, ROC c Department of Microbiology, Soochow University, Shihlin, Taipei, Taiwan, ROC d Department of Biotechnology, Southern Taiwan University, Tainan, Taiwan, ROC ⁎ Corresponding author at: Department of Biochemis Center, 161, Section 6, Min-Chuan East Road, Taipei, Ta 0741; fax: +886 2 8792 3106.. E-mail addresses: gaujang@mail2000.com.tw (K.-C. pslediting@mail.scu.edu.tw (P.-S. Liu), lovelyfish92@yah shch@mail.ndmctsgh.edu.tw (S.-H. Chueh).. 0014-2999/$ – see front matter © 2012 Elsevier B.V. All doi:10.1016/j.ejphar.2012.04.015 a b s t r a c t a r t i c l e i n f o Article history: Received 7 March 2012 Received in revised form 4 April 2012 Accepted 5 April 2012 Available online 20 April 2012 Keywords: Porcine aortic smooth cell Autophagy Endoplasmic reticulum stress Phorbol ester Thapsigargin Extracellular signal-regulated kinase We have previously shown that, in porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stressor thapsigargin simultaneously activate the mitochondrial caspase-dependent death cascade and an extracellular signal-regulated kinase (ERK)-dependent pathway, which inhibits the caspase-independent death pathway. Software:Article Title: Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells. Article Snippet: Chin T-Y, Lin H-C, Kuo J-P, Chueh S-H. Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells.. Am J Physiol Cell Physiol 292: C383–C395, 2007; doi:10.1152/ajpcell.00069.2006.—A sustained increase in the cytosolic Ca concentration ([Ca ]i) can cause cell death.. In this study, we found that, in cultured porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stress, triggered by depletion of Ca stores by thapsigargin (TG), induced an increase in the [Ca ]i and cell death. Article Title: Ultraviolet B irradiation induced Nrf2 degradation occurs via activation of TRPV1 channels in human dermal fibroblasts. Article Snippet: Ultraviolet (UV) irradiation causes cellular oxidative stress.. Under redox imbalance, Keap1-dependent Nrf2 degradation is minimal.. In this study, we examined the role of Ca in Nrf2 homeostasis after UVB irradiation using human dermal fibroblasts. Article Title: Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells. Article Snippet: Contents lists available at SciVerse ScienceDirect European Journal of Pharmacology j ourna l homepage: www.e lsev ie r .com/ locate /e jphar Molecular and Cellular Pharmacology Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells Kao-Chang Lin b,d, Pei-Shan Liu c, Pei-Yu Peng a, Sheau-Huei Chueh a,⁎ a Department of Biochemistry, National Defense Medical Center, Taipei, Taiwan, ROC b Department of Neurology, Chi Mei Medical Center, Tainan, Taiwan, ROC c Department of Microbiology, Soochow University, Shihlin, Taipei, Taiwan, ROC d Department of Biotechnology, Southern Taiwan University, Tainan, Taiwan, ROC ⁎ Corresponding author at: Department of Biochemis Center, 161, Section 6, Min-Chuan East Road, Taipei, Ta 0741; fax: +886 2 8792 3106.. E-mail addresses: gaujang@mail2000.com.tw (K.-C. pslediting@mail.scu.edu.tw (P.-S. Liu), lovelyfish92@yah shch@mail.ndmctsgh.edu.tw (S.-H. Chueh).. 0014-2999/$ – see front matter © 2012 Elsevier B.V. All doi:10.1016/j.ejphar.2012.04.015 a b s t r a c t a r t i c l e i n f o Article history: Received 7 March 2012 Received in revised form 4 April 2012 Accepted 5 April 2012 Available online 20 April 2012 Keywords: Porcine aortic smooth cell Autophagy Endoplasmic reticulum stress Phorbol ester Thapsigargin Extracellular signal-regulated kinase We have previously shown that, in porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stressor thapsigargin simultaneously activate the mitochondrial caspase-dependent death cascade and an extracellular signal-regulated kinase (ERK)-dependent pathway, which inhibits the caspase-independent death pathway. Incubation:Article Title: Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells. Article Snippet: Chin T-Y, Lin H-C, Kuo J-P, Chueh S-H. Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells.. Am J Physiol Cell Physiol 292: C383–C395, 2007; doi:10.1152/ajpcell.00069.2006.—A sustained increase in the cytosolic Ca concentration ([Ca ]i) can cause cell death.. In this study, we found that, in cultured porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stress, triggered by depletion of Ca stores by thapsigargin (TG), induced an increase in the [Ca ]i and cell death. Article Title: Ultraviolet B irradiation induced Nrf2 degradation occurs via activation of TRPV1 channels in human dermal fibroblasts. Article Snippet: Ultraviolet (UV) irradiation causes cellular oxidative stress.. Under redox imbalance, Keap1-dependent Nrf2 degradation is minimal.. In this study, we examined the role of Ca in Nrf2 homeostasis after UVB irradiation using human dermal fibroblasts. Article Title: Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells. Article Snippet: Contents lists available at SciVerse ScienceDirect European Journal of Pharmacology j ourna l homepage: www.e lsev ie r .com/ locate /e jphar Molecular and Cellular Pharmacology Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells Kao-Chang Lin b,d, Pei-Shan Liu c, Pei-Yu Peng a, Sheau-Huei Chueh a,⁎ a Department of Biochemistry, National Defense Medical Center, Taipei, Taiwan, ROC b Department of Neurology, Chi Mei Medical Center, Tainan, Taiwan, ROC c Department of Microbiology, Soochow University, Shihlin, Taipei, Taiwan, ROC d Department of Biotechnology, Southern Taiwan University, Tainan, Taiwan, ROC ⁎ Corresponding author at: Department of Biochemis Center, 161, Section 6, Min-Chuan East Road, Taipei, Ta 0741; fax: +886 2 8792 3106.. E-mail addresses: gaujang@mail2000.com.tw (K.-C. pslediting@mail.scu.edu.tw (P.-S. Liu), lovelyfish92@yah shch@mail.ndmctsgh.edu.tw (S.-H. Chueh).. 0014-2999/$ – see front matter © 2012 Elsevier B.V. All doi:10.1016/j.ejphar.2012.04.015 a b s t r a c t a r t i c l e i n f o Article history: Received 7 March 2012 Received in revised form 4 April 2012 Accepted 5 April 2012 Available online 20 April 2012 Keywords: Porcine aortic smooth cell Autophagy Endoplasmic reticulum stress Phorbol ester Thapsigargin Extracellular signal-regulated kinase We have previously shown that, in porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stressor thapsigargin simultaneously activate the mitochondrial caspase-dependent death cascade and an extracellular signal-regulated kinase (ERK)-dependent pathway, which inhibits the caspase-independent death pathway. Imaging:Article Title: Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells. Article Snippet: Chin T-Y, Lin H-C, Kuo J-P, Chueh S-H. Dual effect of thapsigargin on cell death in porcine aortic smooth muscle cells.. Am J Physiol Cell Physiol 292: C383–C395, 2007; doi:10.1152/ajpcell.00069.2006.—A sustained increase in the cytosolic Ca concentration ([Ca ]i) can cause cell death.. In this study, we found that, in cultured porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stress, triggered by depletion of Ca stores by thapsigargin (TG), induced an increase in the [Ca ]i and cell death. Article Title: Ultraviolet B irradiation induced Nrf2 degradation occurs via activation of TRPV1 channels in human dermal fibroblasts. Article Snippet: Ultraviolet (UV) irradiation causes cellular oxidative stress.. Under redox imbalance, Keap1-dependent Nrf2 degradation is minimal.. In this study, we examined the role of Ca in Nrf2 homeostasis after UVB irradiation using human dermal fibroblasts. Article Title: Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells. Article Snippet: Contents lists available at SciVerse ScienceDirect European Journal of Pharmacology j ourna l homepage: www.e lsev ie r .com/ locate /e jphar Molecular and Cellular Pharmacology Acute phorbol ester treatment inhibits thapsigargin-induced cell death in porcine aortic smooth muscle cells Kao-Chang Lin b,d, Pei-Shan Liu c, Pei-Yu Peng a, Sheau-Huei Chueh a,⁎ a Department of Biochemistry, National Defense Medical Center, Taipei, Taiwan, ROC b Department of Neurology, Chi Mei Medical Center, Tainan, Taiwan, ROC c Department of Microbiology, Soochow University, Shihlin, Taipei, Taiwan, ROC d Department of Biotechnology, Southern Taiwan University, Tainan, Taiwan, ROC ⁎ Corresponding author at: Department of Biochemis Center, 161, Section 6, Min-Chuan East Road, Taipei, Ta 0741; fax: +886 2 8792 3106.. E-mail addresses: gaujang@mail2000.com.tw (K.-C. pslediting@mail.scu.edu.tw (P.-S. Liu), lovelyfish92@yah shch@mail.ndmctsgh.edu.tw (S.-H. Chueh).. 0014-2999/$ – see front matter © 2012 Elsevier B.V. All doi:10.1016/j.ejphar.2012.04.015 a b s t r a c t a r t i c l e i n f o Article history: Received 7 March 2012 Received in revised form 4 April 2012 Accepted 5 April 2012 Available online 20 April 2012 Keywords: Porcine aortic smooth cell Autophagy Endoplasmic reticulum stress Phorbol ester Thapsigargin Extracellular signal-regulated kinase We have previously shown that, in porcine aortic smooth muscle cells, endoplasmic reticulum (ER) stressor thapsigargin simultaneously activate the mitochondrial caspase-dependent death cascade and an extracellular signal-regulated kinase (ERK)-dependent pathway, which inhibits the caspase-independent death pathway. |